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Unravelling roles of error-prone DNA polymerases in shaping cancer genomes | Oncogene
DNA polymerase eta: A potential pharmacological target for cancer therapy - Saha - 2021 - Journal of Cellular Physiology - Wiley Online Library
RCSB PDB - 6CST: Structure of human DNA polymerase kappa with DNA
DNA Polymerase η (E1I7T) Rabbit mAb | Cell Signaling Technology
DNA polymerases and cancer | Nature Reviews Cancer
DNA polymerase eta: A potential pharmacological target for cancer therapy - Saha - 2021 - Journal of Cellular Physiology - Wiley Online Library
Inhibition of Human DNA Polymerases Eta and Kappa by Indole-Derived Molecules Occurs through Distinct Mechanisms | ACS Chemical Biology
A Small-Molecule Inhibitor of Human DNA Polymerase η Potentiates the Effects of Cisplatin in Tumor Cells | Biochemistry
Regulation of the error-prone DNA polymerase Polκ by oncogenic signaling and its contribution to drug resistance | Science Signaling
Genes | Free Full-Text | Maintenance of Genome Integrity: How Mammalian Cells Orchestrate Genome Duplication by Coordinating Replicative and Specialized DNA Polymerases
DNA polymerase eta: A potential pharmacological target for cancer therapy - Saha - 2021 - Journal of Cellular Physiology - Wiley Online Library
Expression of the cancer-associated DNA polymerase ε P286R in fission yeast leads to translesion synthesis polymerase dependent hypermutation and defective DNA replication | PLOS Genetics
DNA polymerase eta: A potential pharmacological target for cancer therapy - Saha - 2021 - Journal of Cellular Physiology - Wiley Online Library
Regulation of the error-prone DNA polymerase polκ by oncogenic signaling and its contribution to drug resistance | bioRxiv
Molecules | Free Full-Text | Mammalian DNA Polymerase Kappa Activity and Specificity
DNA polymerase beta modulates cancer progression via enhancing CDH13 expression by promoter demethylation | Oncogene
PDB-101: Molecule of the Month: Cisplatin and DNA
Inhibition of mutagenic translesion synthesis: A possible strategy for improving chemotherapy? | PLOS Genetics
Switching off DNA repair—how colorectal cancer evades targeted therapies through adaptive mutability | Signal Transduction and Targeted Therapy